Found a microbe that violates the universal principle of DNA

<pre>Found a microbe that violates the universal principle of DNA

A group of researchers from the University of Planck and the Institute of Bath discovered a living creature that, when replicating proteins, violates the basic principle of DNA. Previously, it was believed that the code written in it is by default stable and can only be read in a single way. But with the microorganism Ascoidea asiatica things are different, and he “at will” reads this code in different ways.

DNA molecules are huge and complex, but the principle of reading their code is pretty simple. It is recorded as a sequence of four chemical bases A, C, G and T, and a group of three other bases, called the codon, is used for reading. One codon carries information about one particular amino acid, of which proteins are built, the basic unit of any living cell.

Earlier in the academic world it was believed that it is impossible to read the codon “wrong”, so if you analyze the structure of DNA, you can predict which proteins and in what sequence will be built according to this code. But in the case of the Ascoidea asiatica microbe, the CTG codon in 50% is read as the code for the serine protein, and at other times as leucine. This living creature “cracked” the mechanism of reading its own DNA molecule, and the observer can not predict which protein will be on the way out.

The most interesting thing is that the scientists have not yet understood what benefit the microbe gives. Serin and leucine are very different proteins, simply replacing one another will create a lot of problems for a living organism, and there is a version that such a “lifhak” is not at all necessary to him. Moreover, long-term observations have shown that the microbe tries as little as possible to use the CTG codon, as if he himself is afraid of his strange ability. Then it can be considered as an example of an evolutionary impasse, when the developed super-capacity does not help the creature survive, but it does not disappear due to the death of carriers of a harmful mutation.

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